TMA diagnosis allows the preparation of a diagnostic path, therapeutic and welfare (PDTA) which allows to provide a fundamental contribution in approaching and manage the issue of thrombotic microangiopathy.
The basic aim of this is to know how PDTAs readily recognize patients with a clinical presentation suggestive of TMA, ensuring a correct clinical diagnostic work and above all providing the specific treatment of the disease, be it a hemolytic uremic syndrome, classical or atypical, Thrombotic thrombocytopenia purpura that, in their primary or secondary forms, limiting as much as possible the diagnosis of TMA lost or unrecognized.
The PDTA was drawn schematically to provide a quick reference, useful in diagnostic and practical terms, trying to provide a gradual process to the problem. For this reason we would like to offer a graphic design which includes a basic guide accompanied by numerous schematic references to allow you to orient the operators of a disease that recognizes a similar end effector mechanism, but the pathogenesis currently multifactorial conditioning the clinical manifestations and current therapeutic approaches.
Scientific interest, progressive recent discoveries and the possibility of using new drugs to treat this disease are rapidly changing in this field the clinical-therapeutic scenario will therefore planned upgrades to this first draft.
The basic aim of this is to know how PDTAs readily recognize patients with a clinical presentation suggestive of TMA, ensuring a correct clinical diagnostic work and above all providing the specific treatment of the disease, be it a hemolytic uremic syndrome, classical or atypical, Thrombotic thrombocytopenia purpura that, in their primary or secondary forms, limiting as much as possible the diagnosis of TMA lost or unrecognized.
The PDTA was drawn schematically to provide a quick reference, useful in diagnostic and practical terms, trying to provide a gradual process to the problem. For this reason we would like to offer a graphic design which includes a basic guide accompanied by numerous schematic references to allow you to orient the operators of a disease that recognizes a similar end effector mechanism, but the pathogenesis currently multifactorial conditioning the clinical manifestations and current therapeutic approaches.
Scientific interest, progressive recent discoveries and the possibility of using new drugs to treat this disease are rapidly changing in this field the clinical-therapeutic scenario will therefore planned upgrades to this first draft.
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